單純皰疹病毒1型IgM免疫診斷試劑盒

廣州健侖生物科技有限公司

廣州健侖長期供應(yīng)各種ELISA試劑盒，主要代理進(jìn)口和國產(chǎn)品牌的流行病毒ELISA檢測(cè)試劑盒。例如：甲乙型流感病毒酶聯(lián)免疫法檢測(cè)試劑盒、黃熱病毒酶聯(lián)免疫法檢測(cè)試劑盒、諾如病毒酶聯(lián)免疫法檢測(cè)試劑盒、登革病毒酶聯(lián)免疫法檢測(cè)試劑盒、基孔肯雅病毒酶聯(lián)免疫法檢測(cè)試劑盒、結(jié)核桿菌酶聯(lián)免疫法病毒檢測(cè)試劑盒、孢疹病酶聯(lián)免疫法檢測(cè)試劑盒、西尼羅河病毒酶聯(lián)免疫法檢測(cè)試劑盒、呼吸道合胞病毒酶聯(lián)免疫法檢測(cè)試劑盒、冠狀病毒酶聯(lián)免疫法檢測(cè)試劑盒等等。蟲媒體染病系列、呼吸道病原體系列、發(fā)熱伴出疹系列、消化道及食源感染系列。

檢驗(yàn)原理 單純皰疹病毒1型IgM免疫診斷試劑盒

用抗原包被微量板孔，制成固相載體。加患者血清到板孔中，其所含的抗體特異性地與固相載體中現(xiàn)存抗原結(jié)合，形成免疫復(fù)合物。除去多余物質(zhì)后，加入結(jié)合了堿性磷酸酶的IgG、IgA或IgM抗體，使之與上述免疫復(fù)合物反應(yīng)。洗板，除去多余的結(jié)合物，加入底物（對(duì)硝基苯磷酸鹽）。其與酶結(jié)合的免疫復(fù)合物反應(yīng)，產(chǎn)生有顏色產(chǎn)物，顏色強(qiáng)度與特異性抗體含量成正比。

產(chǎn)品規(guī)格：96T/盒

存儲(chǔ)條件：4-8℃

HSV具有典型皰疹病毒形態(tài)特征。根據(jù)生物化學(xué)、生物學(xué)、流行病學(xué)等分為兩個(gè)血清型，即HSV-1和HSV-2。二者基因組相似，序列有50%的同源性，通過DNA限制性內(nèi)切酶分析來區(qū)分。HSV基因組大約152kb，34個(gè)基因，編碼70多個(gè)多肽（polypeptides）。特別是?基因編碼的晚期蛋白中有11種包膜糖蛋白（gB、gC、gD、gE、gG、gH、gI、gJ、gK、gL、gM），有些功能較清楚。其中g(shù)B和gD與病毒吸附和穿入有關(guān)，是與細(xì)胞特異性受體相互作用的病毒配體分子。gD誘導(dǎo)產(chǎn)生中和抗體的能力zui強(qiáng)，可用于研制疫苗。gC是補(bǔ)體C3b―結(jié)合蛋白（complement C3b-binding protein）。gE是Fc受體，可與IgG的Fc端結(jié)合。gG為型特異性抗原，以此抗原能區(qū)別HSV-1（gG-1）和HSV-2（gG-2）。gH與病毒的釋放有關(guān)。
HSV對(duì)動(dòng)物感染宿主范圍較廣。常用實(shí)驗(yàn)動(dòng)物為家兔、豚鼠及小鼠等。HSV在多種細(xì)胞中能增殖，常用原代兔腎、人胚腎細(xì)胞以及地鼠腎等傳代細(xì)胞培養(yǎng)分離病毒。感染細(xì)胞很快出現(xiàn)明顯細(xì)胞病變，并出現(xiàn)嗜酸性核內(nèi)包涵體。

我司同時(shí)還提供、美國FOCUS、西班牙DIA、美國trinity等試劑盒：

麻疹、風(fēng)疹、甲流 、乙流、單皰疹1型、單皰疹2型、百日咳、百日咳毒素、腮腺炎、帶狀皰疹、單純皰疹、HSV1型特異性、巨細(xì)胞-特異、風(fēng)疹-特異、弓形蟲-特異、棘球?qū)?、嗜肺軍團(tuán)菌、破傷風(fēng)、蜱傳腦炎、幽門螺旋桿菌、白色念珠菌、博氏疏螺旋體、細(xì)小病毒、鉤端螺旋體、腺病毒、Q熱柯克斯體、煙曲霉菌、埃可病毒、EB病毒、衣原體、耶爾森菌、空腸彎曲桿菌、炭疽桿菌、白喉、腸道病毒、柯薩奇病毒、肺炎衣原體、沙眼衣原體、土拉弗朗西斯菌、漢坦病毒、類風(fēng)濕因子、呼吸道合胞病毒、單純皰疹病毒質(zhì)控品、巨細(xì)胞質(zhì)控品、弓形蟲質(zhì)控品、風(fēng)疹麻疹質(zhì)控品、等試劑盒以。

我司還提供其它進(jìn)口或國產(chǎn)試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團(tuán)菌、化妝品檢測(cè)、食品安全檢測(cè)等試劑盒以及日本生研細(xì)菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。

想了解更多的產(chǎn)品及服務(wù)請(qǐng)掃描下方二維碼：

【公司名稱】 廣州健侖生物科技有限公司
【市場部】    楊永漢

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【騰訊  】 2042552662
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-103

海馬回鉤為嗅味覺中樞。顳葉的前部為精 神皮質(zhì)，人類的情緒和精神活動(dòng)不但與眶額皮質(zhì)有關(guān)，與顳葉也大有 關(guān)系，海馬與記憶有關(guān)。顳葉的新皮質(zhì)與額頂枕葉的新皮質(zhì)有纖維相，海馬與基底節(jié)及邊 緣系統(tǒng)有，兩側(cè)顳葉之間也有。聽覺皮質(zhì)區(qū)接受來自內(nèi)側(cè)膝 狀體的沖動(dòng)，通過胼胝體接受來自對(duì)側(cè)顳葉的沖動(dòng)。鉤束繞過外側(cè)裂 顳極和額極。扣帶束有纖維至海馬回，扣帶束前部與鉤束相連， 同時(shí)也接受來自丘腦前核及背側(cè)核、腦干網(wǎng)狀核來的沖動(dòng)。顳葉的傳 出纖維有皮質(zhì)膝狀體束和皮質(zhì)中腦頂蓋束。(1)顳葉癲癇：顳葉病變常 出現(xiàn)癲癇發(fā)作，多表現(xiàn)精神運(yùn)動(dòng)性發(fā)作，可有意識(shí)朦朧、言語錯(cuò)亂、 精神運(yùn)動(dòng)性興奮、定向障礙、情緒紊亂、幻覺、錯(cuò)覺、及記憶缺損等 ，記憶障礙常為發(fā)作的基本癥狀，可有近記憶力、遠(yuǎn)記憶力和現(xiàn)記憶 力障礙，時(shí)間及地點(diǎn)的記憶缺陷明顯。可出現(xiàn)視物變形、變大、變小 等?？捎新牷糜X。自動(dòng)癥是常見表現(xiàn)，發(fā)作時(shí)其活動(dòng)不為意識(shí)所支配 ，可有毀物、傷人、沖動(dòng)、自傷、細(xì)菌、驚恐、發(fā)怒等精神興奮表現(xiàn) ，或出現(xiàn)反復(fù)咀嚼、吞咽、摸索、走動(dòng)等無目的的動(dòng)作?；颊叱Ｓ袎?mèng) 幻覺，顳葉癲癇常有先兆，其中以嗅覺先兆zui常見，幻味發(fā)作也很常 見。(2)記憶障礙：顳葉海馬回破壞時(shí)可出現(xiàn)記憶障礙，通常雙側(cè)損害時(shí)出 現(xiàn)，記憶障礙可伴有定向障礙。(3)聽覺和平衡障礙：一側(cè)損害時(shí)僅有輕度雙側(cè)聽力障礙，雙側(cè)破壞時(shí) 可導(dǎo)致皮質(zhì)性全聾。顳上回也是前庭的皮質(zhì)中樞，細(xì)菌此顳葉病變可 出現(xiàn)平衡障礙和眩暈。(4)言語障礙：顳上回后部損害可出現(xiàn)感覺性失語，患者能聽到講話的 聲音但不能理解其意義，自己的言語也不能聽懂。優(yōu)勢(shì)半球顳上回后 部與頂葉緣上回的移形區(qū)損害時(shí)，可出現(xiàn)命名性失語。(5)視野缺損：顳葉損害時(shí)可出現(xiàn)同向性上1/4象限盲。(6)眼球運(yùn)動(dòng)與瞳孔改變：瞳孔散大，對(duì)光反射消失。如顳葉大面積病 變時(shí)出現(xiàn)小腦幕切跡疝時(shí)出現(xiàn)動(dòng)眼神經(jīng)麻痹。
The hippocampus back to the snoring taste center. Temporal lobe front of the mental cortex, human emotions and mental activity is not only associated with the orbital frontal cortex, and temporal lobe are also greatly related to the memory of the hippocampus. Neocortical temporal lobe and frontal occipital lobe of the new cortex associated with fiber, hippocampus and basal ganglia and the limbic system is linked to the temporal lobe on both sides are also linked. The auditory cortex receives an impulse from the medial geniculate body and receives impulses from the contralateral temporal lobe through the corpus callosum. Hook bundle around the lateral fissure contact temporal pole and the front pole. The cingulate cords have fiber to the hippocampus, the front of the cingulate cords are connected to the hook straps, and the impulses from the anterior thalamic and dorsal nuclei and the brainstem reticular nucleus are also accepted. The temporal lobe of the outgoing fibers of the geniculate geniculate geniculate geniculate bundle and cortex. (1) temporal lobe epilepsy: Temporal lobe lesions often appear epileptic seizures, more performance of psychomotor seizures, may be consciously dim, speech disorders, psychomotor excitation, disorientation, emotional disorders, hallucinations, delusions, and memory impairment, etc., Obstacles are often the basic symptoms of seizures, may have near memory, far memory and memory disorders, time and place of memory defects. There may be visual distortion, larger, smaller and so on. May have hearing hallucinations. Autoimmune is a common manifestation of the activity of the attack is not dominated by consciousness, there may be ruined, wounding, impulsive, self-injury, bacteria, panic, anger and other mental excitement, or repeated chewing, swallowing, groping, walking and so on No purpose of the action. Patients often have fantasy, temporal lobe epilepsy often have aura, of which the most common sense of the smell of the omen, fantasy onset is also very common. (2) memory disorders: temporal hippocampus back damage may occur when memory impairment, usually bilateral damage occurs, memory impairment may be associated with disorientation. (3) auditory and balance disorders: one side of the damage only mild bilateral hearing impairment, bilateral destruction can lead to cortical deafness. The superior temporal gyrus is also the vestibular cortex of the heart, the bacteria can appear in this temporal lobe balance disorders and dizziness. (4) Speech impairment: Sensory aphasia may occur after posterior temporal superiority. The patient can hear the speech but can not understand its meaning, and his speech can not be understood. The dominant hemisphere temporal superior temporal gyrus and parietal lobe edge of the back of the damage zone, there may be a nominal aphasia. (5) visual field defect: temporal lobe damage may appear on the 1/4 quadrant blindness. (6) eye movements and pupil changes: mydriasis, the light reflex disappeared. Such as temporal lobe large lesions appear when the tentorial incision herniation oculomotor nerve paralysis.