WHO可靠血清產(chǎn)品，無(wú)交叉凝集，質(zhì)量保證，反應(yīng)快速，為*優(yōu)質(zhì)血清產(chǎn)品。本司還提供德國(guó)SiFin優(yōu)質(zhì)血清，性價(jià)比高，為各高校實(shí)驗(yàn)室，研究所推薦血清產(chǎn)品！丹麥SSI大腸桿菌血清型鑒定，廣州健侖生物公司提供產(chǎn)品及服務(wù)！A型、B型流感嗜血桿菌多群血清

A型、B型流感嗜血桿菌多群血清

廣州健侖生物科技有限公司

我司還有很多種血清學(xué)診斷血清、血液檢測(cè)、免疫檢測(cè)產(chǎn)品、毒素檢測(cè)、凝集檢測(cè)、酶免檢測(cè)、層析檢測(cè)、免疫熒光檢測(cè)產(chǎn)品，。

（ MOB：楊永漢）

【流感知識(shí)】

流感嗜血桿菌是一種沒(méi)有運(yùn)動(dòng)力的革蘭氏陰性桿菌。它是于1892年由費(fèi)佛博士在流行性感冒的瘟疫中發(fā)現(xiàn)。它一般都是好氧生物，但可以成長(zhǎng)為兼性厭氧生物。
流感嗜血桿菌zui初被誤認(rèn)為是流行性感冒的病因，但直至1933年，當(dāng)發(fā)現(xiàn)流行性感冒的病毒性病原后，才消除了這種誤解。不過(guò)，流感嗜血桿菌仍會(huì)導(dǎo)致其他不同種類的病癥。  

本試劑盒主要用于對(duì)病菌細(xì)菌進(jìn)行檢測(cè)，利用快速玻片凝集檢測(cè)技術(shù)

嗜血桿菌屬血清群A型鑒定

嗜血桿菌屬血清群A型鑒定

嗜血桿菌屬血清群A型鑒定

嗜血桿菌屬血清群A型鑒定

流感嗜血桿菌A/B型凝集抗血清Haemophilus

流感嗜血桿菌A/B型凝集抗血清Haemophilus

流感嗜血桿菌A/B/C型血清群

流感嗜血桿菌A/B/C型血清群

流感嗜血桿菌A/B/C3型凝集抗血清

流感嗜血桿菌A/B/C3型凝集抗血清

a型流感嗜血桿菌診斷血清

a型流感嗜血桿菌診斷血清

玻片凝集法鑒定流感嗜血桿菌

玻片凝集法鑒定流感嗜血桿菌

b型2ml流感嗜血桿菌快速玻片法檢測(cè)血清

b型2ml流感嗜血桿菌快速玻片法檢測(cè)血清

A型、B型流感嗜血桿菌多群血清

我司還提供其它進(jìn)口或國(guó)產(chǎn)試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團(tuán)菌、化妝品檢測(cè)、食品安全檢測(cè)等試劑盒以及日本生研細(xì)菌分型診斷血清、德國(guó)SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。

想了解更多的產(chǎn)品及服務(wù)請(qǐng)掃描下方二維碼：

【公司名稱】 廣州健侖生物科技有限公司
【市場(chǎng)部】    楊永漢

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【騰訊  】 
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號(hào)二期2幢101-103

 

實(shí)驗(yàn)證實(shí)：過(guò)敏人體內(nèi)自由基 數(shù)量比非過(guò)敏人高許多！自由基對(duì)人體免疫系統(tǒng)侵害是過(guò)敏體質(zhì) 形成的基礎(chǔ)，還會(huì)直接氧化人體的肥大細(xì)胞和嗜堿細(xì)胞，導(dǎo)致細(xì) 胞膜破裂釋放出組織胺，產(chǎn)生過(guò)敏反應(yīng)。因此，改善過(guò)敏體質(zhì)就 要清除自由基。自由基來(lái)源于兩個(gè)渠道：一是在機(jī)體本身氧化代 謝過(guò)程中不斷產(chǎn)生；二是環(huán)境污染、輻射、不良生活習(xí)慣等，也 會(huì)不斷產(chǎn)生自由基。自由基可稱萬(wàn)惡之源，百病元兇。人體的老 化、許多疾病的產(chǎn)生都與其密切相關(guān)。自由基所形成的脂質(zhì)過(guò)氧 化物，能夠損害生物膜，破壞細(xì)胞，阻礙正常的新陳代謝，加速 衰老，并能引起諸如免疫失衡，過(guò)敏，頑固皮炎，鼻炎，結(jié)膜炎 ，哮喘，濕疹，高血壓、心肌梗塞、糖尿病、肝炎、痛風(fēng)、腎炎 、白內(nèi)障等多種免疫復(fù)合物是抗體與抗原結(jié)合所得到的一種復(fù)合 物，是由各種免疫細(xì)胞、吞噬細(xì)菌，病毒，致敏物質(zhì)共同死亡后 結(jié)合而形成的，所以又稱抗原--抗體復(fù)合物。它在正常情況下， 小分子可溶性免疫復(fù)合物被腎小球?yàn)V過(guò)排出，大分子不溶免疫復(fù) 合物被巨噬細(xì)胞吞噬消滅，這是機(jī)體防御機(jī)制的一部分。但在某 些情況下，抗原與抗體在體內(nèi)形成的免疫復(fù)合物，沉積于血管壁 基底部，從而激活補(bǔ)體，被激活的補(bǔ)體，發(fā)揮溶解細(xì)菌、病毒、 腫瘤細(xì)胞等作用血清病是由大小免疫復(fù)合物沉積在毛細(xì)血管壁， 補(bǔ)體、吞噬細(xì)胞參與反應(yīng)所致。
The latest authoritative experiments confirmed that the number of free radicals in allergic people is much higher than that of non-allergic people! Free radicals invade the human immune system are the basis for the formation of allergies, but also directly oxidize the body's mast cells and basophils, resulting in rupture of the cell membrane to release histamine, resulting in allergic reactions. Therefore, improving the allergy will eliminate free radicals. Free radicals come from two sources: First, they are constantly generated during the oxidation process of the organism itself; second, environmental pollution, radiation, bad living habits, etc., and free radicals are continuously generated. Free radicals can be called the source of all evil, all the culprits. The aging of the human body and the occurrence of many diseases are closely related to it. The lipid peroxides formed by free radicals can damage biofilms, destroy cells, impede normal metabolism, accelerate aging, and cause diseases such as immune imbalance, allergies, stubborn dermatitis, rhinitis, conjunctivitis, asthma, eczema, and hypertension. Immune complexes such as myocardial infarction, diabetes mellitus, hepatitis, gout, nephritis, and cataracts are complexes obtained by combining antibodies with antigens. They are caused by the death of various immune cells, phagocytosis of bacteria, viruses, and allergens. Combined, it is also called antigen-antibody complex. Under normal conditions, small molecule soluble immune complexes are filtered out by glomerular filtration and macromolecule-insoluble immune complexes are phagocytosed by macrophages, which is part of the body's defense mechanism. But in some cases, immune complexes formed by antigens and antibodies in the body deposit in the basement of the blood vessel wall, which activates complement, activated complement, plays a role in lysing bacteria, viruses, tumor cells, etc. Serum disease is caused by size immunity The complex deposits on the capillary wall and complement and phagocytes participate in the reaction.